Pulmonary Shunt Fraction (Qs/Qt) Calculator

The shunt fraction estimates what percentage of blood passing through the lungs never picks up oxygen at all, quantifying how much of a patient's hypoxemia is due to true shunt versus other causes.

Estimate the Shunt Fraction

g/dL
Used for capillary O₂ content.
Enter a value between 2 and 25.
mmHg
From the alveolar gas equation (A-a gradient step 1).
Enter a value between 20 and 700.
mL/dL
Normal ~16–20 mL/dL.
Enter a value between 3 and 25.
mL/dL
Normal ~14–15 mL/dL.
Enter a value between 2 and 24.
Qs/Qt
15.3%
of pulmonary blood flow
Mildly elevated
CcO₂ (capillary)
20.4 mL/dL
CaO₂ − CvO₂
5.0

How It's Calculated

Formula
Qs/Qt = (CcO₂ − CaO₂) ÷ (CcO₂ − CvO₂)
Capillary content (100% saturated)
CcO₂ = (1.34 × Hgb) + (0.003 × PAO₂)

What the Result Means

< 5%

Physiologic shunt — normal.

5–20%

Mildly elevated.

20–30%

Clinically significant shunt.

> 30%

Severe shunt — refractory to supplemental oxygen.

Worked Example

Given: CcO₂ 20.4, CaO₂ 19.5, CvO₂ 14.5 mL/dL.

  1. Qs/Qt = (20.4 − 19.5) ÷ (20.4 − 14.5) = 0.9 ÷ 5.9
  2. = 0.15 = 15% (mildly elevated).

Blood That Bypasses Gas Exchange

Blood past a collapsed or fluid-filled alveolus stays deoxygenated no matter how much oxygen is given.

ventilated collapsed / filled oxygenated shunted, still deoxygenated

Frequently Asked Questions

Why doesn't more oxygen fix a large shunt?

Shunted blood never contacts ventilated alveoli, so raising FiO₂ can't oxygenate blood that bypasses gas exchange entirely. This refractory hypoxemia is a hallmark of true shunt.

What is a normal shunt fraction?

A physiologic shunt of less than about 5% is normal. Above roughly 20% is clinically significant, and above 30% represents a severe shunt.

What is CcO₂?

The end-capillary oxygen content, calculated assuming the blood leaving a ventilated alveolus is 100% saturated at the alveolar oxygen tension (PAO₂). It's the theoretical best-case content used as the reference.

What conditions cause a large shunt?

ARDS, severe pneumonia, atelectasis, and pulmonary edema all flood or collapse alveoli so blood passes without oxygenating. Structural cardiac shunts do the same at the heart level.

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